NURS 6501: MIDTERM EXAM:
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What is the role of cytochrome C in cellular injury?
Group of answer choices
- Cytochrome C promotes the production of reactive oxygen species.
- Cytochrome C is released into the cytoplasm to enhance cellular repair mechanisms.
- Cytochrome C promotes apoptosome formation and apoptosis.
- Cytochrome C interacts with mitochondrial DNA to mitigate damage during injury.
- Cytochrome C promotes apoptosome formation and apoptosis: Cytochrome C is a key protein located in the mitochondria. During cellular stress or damage, it is released into the cytoplasm, where it interacts with other proteins (such as Apaf-1) to form the apoptosome. This complex activates caspases, which are enzymes responsible for carrying out the apoptotic process (programmed cell death).
- Cytochrome C promotes the production of reactive oxygen species: While mitochondria are involved in generating reactive oxygen species (ROS), cytochrome C itself is not directly responsible for their production. ROS generation is more closely related to mitochondrial dysfunction or oxidative stress, not cytochrome C's role in apoptosis.
- Cytochrome C is released into the cytoplasm to enhance cellular repair mechanisms: Cytochrome C's release is associated with apoptosis, not repair mechanisms. Its release triggers cell death, not repair processes.
- Cytochrome C interacts with mitochondrial DNA to mitigate damage during injury: Cytochrome C does not interact with mitochondrial DNA to mitigate damage. Its main function in cellular injury is to initiate apoptosis when cellular damage is beyond repair.